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Paroxysmal cold hemoglobinuria

In the inflammatory muscle disease polymyocystitis autoantibodies are often directed against cytoplasmic proteins including aminoacyl-tRNA synthetases. In the rare paroxysmal cold hemoglobinuria autoantibodies attack red blood cell membranes only when the temperature of an extremity is lowered. Paroxysmal nocturnal hemoglobinuria, a serious complement-mediated condition, results from deficiency in the complement decay accelerating factor. This is a result of a defect in the PGI tail on this factor. ... [Pg.952]


See other pages where Paroxysmal cold hemoglobinuria is mentioned: [Pg.1865]    [Pg.1865]    [Pg.471]    [Pg.2208]    [Pg.271]    [Pg.952]    [Pg.931]    [Pg.420]    [Pg.1865]    [Pg.1865]    [Pg.471]    [Pg.2208]    [Pg.271]    [Pg.952]    [Pg.931]    [Pg.420]    [Pg.251]    [Pg.172]   
See also in sourсe #XX -- [ Pg.1865 ]




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