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Pacing systems modes

Sinus node dysfunction was initially described in the early 1900s, and is the primary indication for pacemaker implantation in industrialized countries. The only effective treatment for symptomatic sinus node dysfunction is cardiac pacing. However, despite the widespread use of pacing therapy for this group of patients, the optimal pacing mode, pacing system and site of ventricular stimulation for sinus node dysfunction remains controversial. The available data for the diagnosis and treatment of sinus node dysfunction are reviewed in this chapter. [Pg.377]

Pacing system inhibition from interference is more important in the pacemaker-dependent patient. If inhibition is observed in a pacemaker-dependent patient, electrocautery application should be limited to 1-2 s with a rest period of approximately 10 s. This will allow the pacemaker to function properly for a greater portion of the time. In some cases back-up temporary transvenous or transcutaneous pacing will be required, or the pacemaker can be programmed to the asynchronous mode of function. [Pg.599]

Reinterrogate the pacing system after defibrillatation or cardioversion. A programmed parameter, such as a mode of function, should be programmed to confirm that the pacemaker actually performs telemetered instructions. [Pg.601]

Bio-control using pathogens holds promise mostly in non-cropland situations because of the slow pace of control of weeds and the wider window for control as compared with the shorter window of the cropping season and associated disturbances under cropland situations. However, in aquatic systems they appear to be more realistic, due to the absence of such cropping barriers and enhanced mode of dispersal in water. Among the wetland weeds, Echinochloa crusgalli and Cyperus rotundas are being... [Pg.110]

The models most frequently used to describe the concentration dependence of diffusion and permeability coefficients of gases and vapors, including hydrocarbons, are transport model of dual-mode sorption (which is usually used to describe diffusion and permeation in polymer glasses) as well as its various modifications molecular models analyzing the relation of diffusion coefficients to the movement of penetrant molecules and the effect of intermolecular forces on these processes and free volume models describing the relation of diffusion coefficients and fractional free volume of the system. Molecular models and free volume models are commonly used to describe diffusion in rubbery polymers. However, some versions of these models that fall into both classification groups have been used for both mbbery and glassy polymers. These are the models by Pace-Datyner and Duda-Vrentas [7,29,30]. [Pg.240]

Fig. 10.5 Sinus rhythm with second-degree Type 13 2 iniranodal AV block, and RBBB. Note that the AH interval remains constant. TTie HV interval increases from 80 (following first P-wave) to 150ms (following second P-wave). The third P-wave is followed by an H deflection but no QRS complex. AV block occurs in the His-Purkinje system below the site of recording of the His bundle potential (arrow). Note the shorter PR interval after the nonconducted P-wave, a feature typical of Type I second-degree AV block. HBE = His bundle electrogram, A = atrial deflection, H = His bundle deflection, V = ventricular deflection, P = P-wave. TL = time lines 50ms. (Barold SS. Pacemaker treatment of bradycardias and selection of optimal pacing modes. In Zipes DP (Ed.). Contemporary Treatments in Cardiovascular Disease, 1997 1 123, with pamission.)... Fig. 10.5 Sinus rhythm with second-degree Type 13 2 iniranodal AV block, and RBBB. Note that the AH interval remains constant. TTie HV interval increases from 80 (following first P-wave) to 150ms (following second P-wave). The third P-wave is followed by an H deflection but no QRS complex. AV block occurs in the His-Purkinje system below the site of recording of the His bundle potential (arrow). Note the shorter PR interval after the nonconducted P-wave, a feature typical of Type I second-degree AV block. HBE = His bundle electrogram, A = atrial deflection, H = His bundle deflection, V = ventricular deflection, P = P-wave. TL = time lines 50ms. (Barold SS. Pacemaker treatment of bradycardias and selection of optimal pacing modes. In Zipes DP (Ed.). Contemporary Treatments in Cardiovascular Disease, 1997 1 123, with pamission.)...
Fig. 19.12 Event histogram from a patient implanted with a Trilogy DR+. The indication for pacing was the bradycardia-tachycardia syndrome. Over the past 42 days, the majority of the complexes were in the base rate or sleep rate bins. Rate modulation had not yet been enabled and there were relatively few native atrial rates above 75 bpm. The system functioned in a nontracking mode approximately 3% of the time (AMS bin) with 1,776 mode switch episodes. Based on this event histogram demonstrating the presence of chronotropic incompetence, rate-modulation was enabled. Fig. 19.12 Event histogram from a patient implanted with a Trilogy DR+. The indication for pacing was the bradycardia-tachycardia syndrome. Over the past 42 days, the majority of the complexes were in the base rate or sleep rate bins. Rate modulation had not yet been enabled and there were relatively few native atrial rates above 75 bpm. The system functioned in a nontracking mode approximately 3% of the time (AMS bin) with 1,776 mode switch episodes. Based on this event histogram demonstrating the presence of chronotropic incompetence, rate-modulation was enabled.

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