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Non receptor triggers reactive oxygen species and nitric oxide

Non receptor triggers reactive oxygen species and nitric oxide [Pg.36]

Mitochondria are the site of ROS production during ischemia. Arachidonic acid, eNOS, NADPH oxidase and xanthine oxidase are sources of reactive oxygen species at reperfusion. Cytochrome P450 monooxygenases (CYPs) have also been implicated. CYP catalyzes arachidonic acid oxidation to a variety of biologically active eicosanoids and generates reactive oxygen species.1 3 [Pg.37]

Paradoxically, ROS are not only injurious but may also be protective. ROS are shown to upregulate survival signaling.119 Furthermore, the cardioprotective abilities of ischemic preconditioning are completely abolished with the administration of N-acetylcysteine indicating that ROS can act as important mediators of the preconditioning response.120 (See also chapter 3). [Pg.38]




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Nitric oxide oxygenation

Nitric oxide reactivity

Non species

Non-oxidative

Non-reactive

Non-reactive species

Oxidation species

Oxygen and oxidation

Oxygen species

Oxygenated species

Reactive oxidants

Reactive oxygen

Reactive oxygen reactivity

Reactive oxygen species

Reactive oxygen species , nitric oxide

Reactive oxygen species and

Reactive species

Reactive species reactivity

Receptors reactivation

Triggerable

Triggers

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