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Lung disease surface area

In general, it is recognized that there are multiple cellular and molecular responses to asbestos fibers, that no single mechanism is likely to account for all asbestos-related diseases, that the precise steps in pathogenic pathways leading to asbestos-related disease are not fiilly established, and that fiber structural and chemical properties (e.g., length, width, iron content, durability, surface areas) are important variables that play a role in the development of lung and pleural injury. [Pg.411]

Brown DM, Wilson MR, MacNee W, Stone V, Donaldson K (2001) Size-dependent proinflammatory effects of ultrafine polystyrene particles a role for surface area and oxidative stress in the enhanced activity of ultrafines. Toxicol Appl Pharmacol 175 191-199 Brunkreef B, Holgate ST (2002) Air pollution and health. Lancet 360 1233-1242 Chen H, Goldberg MS, Villeneuve PJ (2008) A systematic review of the relation between longterm exposure to ambient air pollution and chronic diseases. Rev Environ Health 23 243-297 Churg A, Brauer M (1997) Human lung parenchyma retains PM2.5. Am J Respir Crit Care Med 155 2109-2111... [Pg.424]

See other pages where Lung disease surface area is mentioned: [Pg.398]    [Pg.295]    [Pg.258]    [Pg.550]    [Pg.288]    [Pg.257]    [Pg.165]    [Pg.166]    [Pg.997]    [Pg.2249]    [Pg.2262]    [Pg.82]    [Pg.1621]    [Pg.103]    [Pg.89]    [Pg.102]    [Pg.592]    [Pg.749]    [Pg.757]    [Pg.122]    [Pg.54]    [Pg.77]    [Pg.378]    [Pg.247]    [Pg.153]    [Pg.367]    [Pg.245]    [Pg.129]    [Pg.122]    [Pg.305]    [Pg.1211]    [Pg.349]    [Pg.18]    [Pg.254]    [Pg.71]    [Pg.390]    [Pg.2268]    [Pg.150]    [Pg.2194]    [Pg.243]    [Pg.3148]    [Pg.3442]    [Pg.8]    [Pg.164]    [Pg.257]   
See also in sourсe #XX -- [ Pg.73 ]



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Lungs surface area

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