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Ischemic contracture

Herse DJ, Garlick PB, Humphrey SM (1977) Ischemic contracture of the myocardiiun mechanisms and prevention. Am J Cardiol 39 986-993... [Pg.320]

K.G. Kolocassides, M. Galinanes and D.J. Hearse, Dichotomy of ischemic preconditioning. Improved postischemic contractile function despite intensification of ischemic contracture, Circulation 93, 1725— 1733(1996). [Pg.67]

Figure 3. Lcll ventricular pressure recordings obtained from isolated rat hearts subjected to zero-flow global ischemia and reperfusion. Ischemic contracture is exacerbated in thyroxine treated hearts (13) and is suppressed in the hypothyroid hearts (C) as compared to normal hearts (A). Recovery of left ventricular developed pressure is increased in both hypothyroid and hyperthyroid as compared to normal hearts. LVDP is defined as the difference between systolic and diastolic left ventricular pressure (data from our laboratory). Figure 3. Lcll ventricular pressure recordings obtained from isolated rat hearts subjected to zero-flow global ischemia and reperfusion. Ischemic contracture is exacerbated in thyroxine treated hearts (13) and is suppressed in the hypothyroid hearts (C) as compared to normal hearts (A). Recovery of left ventricular developed pressure is increased in both hypothyroid and hyperthyroid as compared to normal hearts. LVDP is defined as the difference between systolic and diastolic left ventricular pressure (data from our laboratory).
C. Pantos, D.D. Cokkinos, S. Tzeis, V. Malliopoulou, I. Mourouzis, H. Caragcorgiou, C. Limas, D. Varonos and D.V. Cokkinos, Hyperthyroidism is associated with preserved preconditioning capacity but intensified and accelerated ischemic contracture in rat heart, Basic Res. Cardiol. 94,254-260 (1999). [Pg.97]

In isolated perfused hearts, ventricular dysfunction may be due to myocardial stunning or lethal cell injury. In the Langendorff perfused ischemic rat hearts, ATP concentrations decrease rapidly to 60% in the first minute, with a rapid secondary decrease by 13 min due to contracture (33). Recovery from stunned to normal myocardium requires 24-48 h in the in vivo reperfused heart with coronary artery occlusion of 2-20 min (34). Dobrinina et al. (11) showed that neutral liposomes preserved liver integrity in rats subjected to hepatotropic poisons by non-specific mechanisms. However, histochemical infarct size data do not support this hypothesis in the myocardium (Fig. 3b). [Pg.317]

N.B. Butwell, R. Ramasamy, I. Lazar, A.D. Sherry, and C.R. Malloy, Effect of Lidocaine on Contracture, Intracellular Sodium, and pH in Ischemic Rat Hearts. Am. J. Physiol, 264(6Pt2), H1884-H1889,1993. [Pg.324]

See other pages where Ischemic contracture is mentioned: [Pg.94]    [Pg.374]    [Pg.14]    [Pg.20]    [Pg.20]    [Pg.22]    [Pg.23]    [Pg.29]    [Pg.54]    [Pg.87]    [Pg.88]    [Pg.91]    [Pg.177]    [Pg.177]    [Pg.187]    [Pg.94]    [Pg.374]    [Pg.14]    [Pg.20]    [Pg.20]    [Pg.22]    [Pg.23]    [Pg.29]    [Pg.54]    [Pg.87]    [Pg.88]    [Pg.91]    [Pg.177]    [Pg.177]    [Pg.187]    [Pg.697]    [Pg.702]    [Pg.22]    [Pg.172]    [Pg.69]   
See also in sourсe #XX -- [ Pg.14 , Pg.20 , Pg.177 ]



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