Infarction with disappearing

Of note, an interesting phenomenon that sometimes occurs in the subacute phase of stoke is the CT fogging effect, in which irreversibly infarcted regions disappear on unenhanced CT performed on older generation scanners, 2-6 weeks after stroke onset (Fig. 3.3). With fogging, the acute-phase CT low attenuation - due to edema - is thought to resolve, to be replaced by higher attenuation infiltration of... 

Some patients with acute myocardial infarction (AMI) are found to have HIT antibodies career state of HIT antibodies regardless of previous heparin usage. In those patients, early-onset HIT has been observed, which occurs within a very short period after UFH administration during PCI even if it is the initial exposure to UFH (10). Once HIT antibodies are generated after the exposure to UFH, the antibodies do not disappear for approximately 100 days after the cessation of UFH. If UFH intervention is resumed while the antibodies remain, HIT may readily develop as rapid-onset type of HIT (II). Thrombotic complications are highly anticipated following the abrupt onset of HIT in patients who have been exposed recently to UFH. 

Worsening of angina pectoris has been attributed to beta-blocker therapy. The reports include 35 cases in a series of 296 elderly patients admitted to hospital with suspected myocardial infarction in these 35 the pain disappeared within 7 hours of withdrawing beta-blocker therapy (60). 

Figure 8.12 Patient with extensive anterior infarction. ECG normalisation with Q wave disappearing and positivation of T wave during 18-month follow-up.

The plaques of Alzheimer s disease and the fibrous state of the prions of mad cow disease (both with resulting brain destruction), the thrombi of stroke (cerebral thrombosis) and of heart attack (myocardial infarction), and the familiar manifestation of death (rigor mortis) represent excursions too far in the direction of protein insolubility. The favorable actions of antioxidants keep proteins from becoming so soluble (unfolded) that protein function disappears and proteolytic degradation ensues. Of course, the lack of blood clotting, hemophilia (the lack of clotting proteins to become insoluble by association of oil-like domains), results in death. Such devastations result from loss of proper balance between solubility and insolubility. They represent excursions too far from the cusp of insolubility, that is, too far from the boundary between insolubility and solubility. 

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