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Genetic resistance antibodies

TNFa is thought to be an important mediator in the suppression of CHS. This hypothesis is supported by the finding that injection of antibodies to TNFa reverses the suppression of CHS that usually occurs following UVR exposure. Conversely, intracutaneous injection of recombinant TNFa at the site of sensitization suppresses the development of a contact sensitivity response. Also, the genetic difference between UVR-resistant and UVR-susceptible strains of mice appears to involve a polymorphism in a regulatory region of the TNFa gene. [Pg.783]

See other pages where Genetic resistance antibodies is mentioned: [Pg.311]    [Pg.18]    [Pg.97]    [Pg.499]    [Pg.530]    [Pg.233]    [Pg.128]    [Pg.11]    [Pg.366]    [Pg.203]    [Pg.108]    [Pg.340]    [Pg.464]    [Pg.476]    [Pg.131]    [Pg.1696]    [Pg.1249]    [Pg.2]    [Pg.125]    [Pg.44]    [Pg.351]    [Pg.137]    [Pg.102]    [Pg.233]    [Pg.797]    [Pg.132]    [Pg.75]    [Pg.461]    [Pg.686]    [Pg.185]    [Pg.355]    [Pg.66]    [Pg.692]    [Pg.803]    [Pg.251]    [Pg.1]    [Pg.189]    [Pg.306]    [Pg.1502]    [Pg.266]    [Pg.193]    [Pg.91]    [Pg.110]    [Pg.728]    [Pg.15]    [Pg.131]    [Pg.42]   
See also in sourсe #XX -- [ Pg.131 , Pg.136 ]



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