Deletion hypothesis

This competitive recruitment hypothesis explains how TNF-R2, a survival receptor, could induce apoptosis (Fig. 12B). It has been observed that mice deficient in TNF-R2 showed decreased cell death following TNF treatment (Erickson et at., 1994), while overexpression of TNF-R2 could lead to increased sensitivity to TNF-induced apoptosis (Chan and Lenar-do, 2000 Haridas et at., 1998 Heller et at., 1992 Vandenabeele et at, 1995 Weiss et at., 1997). Moreover, TNF-induced apoptosis of activated primary T lymphocytes has also been shown to require TNF-R2 (Sarin et at., 1995 Zheng et at., 1995). Functionally, this may be important for Fas-indepen-dent peripheral deletion of T lymphocytes and the regulation of mature T-cell homeostasis. 

Inspection of the fabric attribute dimensions for all location points revealed that certain attributes were either absent or unrecognizable. These attributes are fiber bundle, fiber single, fiber crimp, fiber surface, fiber pattern, yarn type, and yarn twist direction. Therefore, these attributes were deleted from the statistical analysis. The remaining attributes were either present at all location points or exhibited variation. These attributes included the three (layers, 1/1, and float) whose interactive effects had direct relevance to the research hypothesis, and the following fiber and yarn attributes paired fibers, 0 twist, combined yarns, yarn system A, yarn systems A + B, yarn systems A + B + C + D, fabric distortion, red, green, and black. 

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