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Angiotensin acute cyclosporine nephrotox

The interaction of cyclosporine with the plasma and tissue renin-angiotensin-aldosterone systems (RAS) has been extensively studied [21, 22]. Sodium depletion, a condition that stimulates renin release, enhances acute CSA nephrotoxicity [23, 24]. In rats, CSA treatment enhanced plasma renin activity (PRA) [21, 25, 26], increased renal renin content [21], promoted juxtaglomerular hypertrophy and hyperplasia [27,... [Pg.619]

Prevot A, Semama DS, Tendron A, Justrabo E, Guignard JP, Gouyon JB. Endothelin, angiotensin II and adenosine in acute cyclosporine A nephrotoxicity. Pediatr Nephrol 2000 14 927-934. [Pg.652]

Grieve EM, Hawksworth GM, Simpson JG, Whiting PH. Effect of thromboxane synthetase inhibition and angiotensin converting enzyme inhibition on acute cyclosporin A nephrotoxicity. Biochem Pharmacol 1990 40 2323-2329. [Pg.652]

See other pages where Angiotensin acute cyclosporine nephrotox is mentioned: [Pg.175]    [Pg.78]   
See also in sourсe #XX -- [ Pg.405 ]



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Cyclosporine nephrotoxicity

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Cyclosporins Cyclosporin

Nephrotoxicity

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