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T amplitude by age in lead aVR, each curve corresponding to the indicated

T amplitude by age in lead I, each curve corresponding to the indicated percentile level . T waves in lead I are small, absent, or even negative, shortly after birth and their amplitude Increases gradually, reaching a plateau by age 3-6 months, and stays little changed until adolescence.

T amplitude by age in lead II, each curve corresponding to the Indicated percentile level . T waves are small after birth. The T waves amplitude Increases rapidly after age 3-7 days to age 3-6 months, then gradually thereafter up to 3-6 years of age. It decreases slightly after age 9-13 years until age 13-18 years.

T amplitude by age in lead III, each curve corresponding to the Indicated percentile level . T waves changes in lead III during the newborn period are very characteristic. The T amplitude decreases even becomes negative, reaching a nadir at age 3-7 days. It increases thereafter, up to a plateau by age 6-12 months until adolescence.

T amplitude by age in lead V1, each curve corresponding to the indicated percentile level . T waves in VI are characteristically upright, low or only slightly Inverted during the first day of life. They all become inverted with increased depth until age 3-6 months, then turning to be less inverted or even upright thereafter along with increasing age.

T amplitude by age in lead V2, each curve corresponding to the indicated percentile level . T wave changes by age in lead V2 are not as spectacular and characteristic as those registered In lead VI.

T amplitude by age in lead V4, each curve corresponding to the indicated

T amplitude by age in lead V5, each curve corresponding to the indicated percentile level . The T waves changes recorded in V5 from birth to adolescence are similar to those recorded In V4, except that the decrease of the T waves amplitude after age 9-13 years are more prominent.

T amplitude by age in lead V6, each curve corresponding to the indicated percentile level . Normal T wave changes registered in lead V6 are more striking and characteristic than those registered In V5.

T and a dependence of conductivity a Tempera-

T and T2 maps of the olfactory bulb of a young rabbit reconstructed directly from the noisy experimental images . For Tx mapping, the recovery curve of a Spin-Echo sequence was sampled at n 8 different repetition times, x 500, 1000, 1500, 2000, 2500, 3000, 4000, 5000 T. For T2 mapping, the decay curve of the same Spin-Echo sequence was sampled at n 5 different echo times, x 10, 35, 60, 85, 110 T. For both, the model considers a single exponential term and the reconstruction is performed using LS approach.

T Animations helpful in learning reaction mechanisms included in The Interesting Chemistry CDs

T as a function of N-type crosslinks

T as a function of PEG content. The numbers in the figure indicate different molecular weights of PEG.

T as a function of Q tor R — 0.1 . The parameters characterizing the system are do —222s , b—739s V , y —5070 s, and 0-0.054 s Note that the abscissa scale is so greatly contracted as to make it impossible to draw the decreasing behavior of T with increasing Q which would be exhibited in the weak-Q region.

T bonding in a complex.

T bound state of the square-well potential for values of R 10,20,..., 100.

T C column shear vs. drift

T C failure mode

T C values for in vivo xenograft mouse models using the human cell lines PC3, CAKI-1, A431 and MCF-7 in connection with titanocenes 3 and 14

T cell-mediated neuroprotection in a PD model. In MPTP-intoxicated mice, regnlatory T cells infiltrate the inflamed nigrostriatal pathway where they encounter cross-reactive CNS antigens presented in the context of MHC by resident microglial cells. In response, activated T cells secrete anti-inflammatory cytokines such as IL, IL-10, and TGF-P that suppress toxic microglial activities. Neurotrophin expression may occur directly from T cells or T ceU-derived IL-4, and IL-10 may induce neurotrophin production in neighboring glia. These activities lead to neuroprotection indirectly by suppression of microglial responses and directly through the local delivery of neurotrophins.

T chemical shift values of two cobalt complexes with the phosphinidenes in trans apexes of a Co P octahedron

T combustion instability evaluated on the basis of the m n stability criterion.

T Comparative kinetic studies for reduction with 163 PFf and BARF salts ,

T concentration decay during the same treatments of



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