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Satiety signals

Cholecystokinin (CCK) is produced in the intestine and the brain. It appears to be an important mediator of anxiety. It also stimulates vasopressin secretion and slows gastric emptying. In addition, it is an important humoral satiety signal (appetite control). Various antagonists have been developed and are currently being investigated with regard to their therapeutic potential. [Pg.356]

Smith, G. P. and J. Gibbs (1975). Cholecystokinin a putative satiety signal. Pharmacol Biochem Behav 3(Suppl 1) 135-8. [Pg.16]

Nucleus Tractus Solitaris (NTS) and Peripheral Satiety Signals, 873... [Pg.838]

Harvey J, Ashford ML. Leptin in the CNS Much more than a satiety signal. Neuropharmacology 2003 44 845-854. [Pg.2675]

See other pages where Satiety signals is mentioned: [Pg.209]    [Pg.209]    [Pg.209]    [Pg.210]    [Pg.212]    [Pg.374]    [Pg.229]    [Pg.351]    [Pg.544]    [Pg.209]    [Pg.209]    [Pg.209]    [Pg.210]    [Pg.212]    [Pg.552]    [Pg.381]    [Pg.381]    [Pg.981]    [Pg.381]    [Pg.381]    [Pg.11]    [Pg.13]    [Pg.13]    [Pg.869]    [Pg.873]    [Pg.873]    [Pg.886]    [Pg.1874]    [Pg.509]    [Pg.616]    [Pg.266]    [Pg.150]    [Pg.491]    [Pg.491]   
See also in sourсe #XX -- [ Pg.6 , Pg.873 ]



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