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Reperfusion injury, oxygen radical-nitric

The free radical superoxide, O2, reacts with nitric oxide, NO, to produce damaging peroxinitrite, OONO . This process has been postulated to be a mediator of ischemia-reperfusion injury as well as inflammatory and vascular diseases. Superoxide dismutases (SODs), enzymes discussed previously, are responsible for converting superoxide to hydrogen peroxide and oxygen. SODs are... [Pg.47]

See other pages where Reperfusion injury, oxygen radical-nitric is mentioned: [Pg.98]    [Pg.364]    [Pg.182]    [Pg.64]    [Pg.149]    [Pg.157]    [Pg.448]    [Pg.164]    [Pg.414]   


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Reperfusion

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