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Regulation of TAG Mobilization

Fig. 11.6 (Hormonal) regulation of TAG mobilization in adipocytes by translocation of HSL and perilipin as well as the molecular mechanisms involved (phosphorylation by PKA, dephosphorylation by protein phospha-... Fig. 11.6 (Hormonal) regulation of TAG mobilization in adipocytes by translocation of HSL and perilipin as well as the molecular mechanisms involved (phosphorylation by PKA, dephosphorylation by protein phospha-...
Tab. 11.2 Differences in the regulation of TAG mobilization and storage between fat cells from visceral (vAT) and subcutaneous adipose tissue (scAT). The extent of some of these differences depends on the adipocyte hyperplasie (total fat mass) and hypertrophy as well as on the sex. Tab. 11.2 Differences in the regulation of TAG mobilization and storage between fat cells from visceral (vAT) and subcutaneous adipose tissue (scAT). The extent of some of these differences depends on the adipocyte hyperplasie (total fat mass) and hypertrophy as well as on the sex.
Tab. 11.1 Natural and synthetic ligands known to affect TAG storage and/or mobilization in WAT via receptor-mediated mechanisms. Meanwhile, the adipocyte has gained the status of an endocrine cell with the discovery of regulated production of several secreted products of various nature (e.g. lipid metabolites, cytokines and other proteins) that are involved in autocrine, paracrine and endocrine effects on TAG storage and mobilization. Adapted from Ref. [559] with modifications. Tab. 11.1 Natural and synthetic ligands known to affect TAG storage and/or mobilization in WAT via receptor-mediated mechanisms. Meanwhile, the adipocyte has gained the status of an endocrine cell with the discovery of regulated production of several secreted products of various nature (e.g. lipid metabolites, cytokines and other proteins) that are involved in autocrine, paracrine and endocrine effects on TAG storage and mobilization. Adapted from Ref. [559] with modifications.
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