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Damage distribution and mean stress

Figure 2. Damage distribution and mean stress contour plot at 0.6 ms in computer simulation of Experiment 79-8. The contour level and plot dimensions are the same as in Figure 1. At this time, the detonation is complete. A shock wave is propagating upward toward the free surface. Extensive damage has occurred around the... Figure 2. Damage distribution and mean stress contour plot at 0.6 ms in computer simulation of Experiment 79-8. The contour level and plot dimensions are the same as in Figure 1. At this time, the detonation is complete. A shock wave is propagating upward toward the free surface. Extensive damage has occurred around the...
A more common method for medical devices is to run the life test until failure occurs. Then an exponential model can be used to calculate the percentage survivability. Using a chi-square distribution, limits of confidence on this calculation can be established. These calculations assume that a failure is equally likely to occur at any time. If this assumption is unreasonable (e.g., if there are a number of early failures), it may be necessary to use a Weibull model to calculate the mean time to failure. This statistical model requires the determination of two parameters and is much more difficult to apply to a test that some devices survived. In the heart-valve industry, lifetime prediction based on S-N (stress versus number of cycles) or damage-tolerant approaches is required. These methods require fatigue testing and ability to predict crack growth. " ... [Pg.336]

Thick-layer elastic-bonded joints first yield before they tear when subjected to peeling forces, allowing the load to be distributed over a wider area. Consequently, the stresses within the bonded materials are kept at a relatively low level. The high tear propagation strength of polyurethanes - even after tear initiation - usually prevents sudden and catastrophic failure of the joint. This yielding behaviour means that damaged adhesive joints can often be identified and repaired before... [Pg.374]

There are several possible reasons why the relationship between the spatial extent of forest damage and the distribution of air pollution might be obscured. The actual dose of specific pollutants received by trees growing in forests is very difficult to assess and figures are generally taken from area-averaged models. This can sometimes result in a considerable underestimate of the pollution load (Fowler et ai. 1989). Another problem is that pollution may only be important at sites already stressed by other factors. This means that a specific pollutant load may have different effects in different circumstances an effect that will invalidate any standard regression analysis. [Pg.65]


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