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Conversions isoform interactions

According to the protein-only hypothesis, proposed by Prusiner (Fig. 5), the interaction of the cellular prion protein with the pathological isoform seems to be the crucial step in the conversion of PrP = to PrP . The existence of the hypothetical PrP =/PrP heterodimer may require the presence of a homodimer consisting of two PrP = molecules. This homodimer is thought to be in equilibrium with the PrP monomers. It is unclear to date wether the spontaneous conversion reaction involves PrP monomers or the PrP homodimers. [Pg.248]

See other pages where Conversions isoform interactions is mentioned: [Pg.916]    [Pg.255]    [Pg.257]    [Pg.349]    [Pg.310]    [Pg.38]    [Pg.252]    [Pg.175]    [Pg.335]    [Pg.154]    [Pg.221]    [Pg.145]    [Pg.1742]    [Pg.634]    [Pg.23]    [Pg.225]    [Pg.361]    [Pg.139]    [Pg.141]    [Pg.143]    [Pg.145]    [Pg.147]    [Pg.149]    [Pg.151]    [Pg.153]    [Pg.155]    [Pg.157]    [Pg.159]    [Pg.163]    [Pg.165]    [Pg.167]    [Pg.169]    [Pg.203]    [Pg.231]    [Pg.205]    [Pg.236]    [Pg.645]    [Pg.669]    [Pg.26]    [Pg.117]    [Pg.395]    [Pg.408]    [Pg.399]    [Pg.320]    [Pg.49]    [Pg.309]    [Pg.317]   
See also in sourсe #XX -- [ Pg.248 , Pg.249 , Pg.250 ]



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